Organoids for Further Research


     Organoids are tissues that resemble human’s “eye, gut, liver, kidney pancreas, prostrate, lung, stomach, and breast”, produced by scientists.  Since they are of great similarity in structure and function to human tissues, organoids are being used to increase knowledge in diseases and drugs. They are small enough to live without any blood, but big enough to study its development and physiology. Individual cell cultures are different from organoids in that they are grown flat and within just one specific cell. Organoids are more complex than that because they require a mix of progenitors and cells to able to grow in a 3-D structure. They cannot show the affects of a therapy on an organism. In the in vivo research, one can see how certain things affect the immune system of the animal. 

     Matrigel is a gelatinous substance that is kind of like the extracellular matrix. Matrigel is critical for growing organoids so that they will grow in a 3-D structure rather than flat. In particular they have been used to study the effectiveness of a drug for cystic fibrosis. If successful, the drug will open the ion channels so that water could flow inward. Scientist also use organoids to find the best therapies for patients with cancer, by using drugs to test them on specific tumors. They can convert organoids into cancer to resemble the tumors and then use them for studies to be able to develop therapies. This can lead to organogenesis in the future because if a patient needs an organ, they can basically grow it for them and just implant it. This would save the trouble of having to be put on a wait list for a perfect match, and in addition to that there is less possibility that the body will reject it.


 Transforming normal organoids into cancer 

These neural stem cells and neurons make a brain organoid

References:

Dekkers, Johanna F. “A Functional CFTR Assay Using Primary Cystic Fibrosis Intestinal Organoids.” Nature Medicine, 6 Oct. 2017, www.nature.com/nm/journal/v19/n7/full/nm.3201.html.  
 


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